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Myofiber-specific TEAD1 overexpression drives satellite cell hyperplasia and counters pathological effects of dystrophin deficiency
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5059137/
Super-numeral SCs attain a ‘normal’ quiescent state, accelerate regeneration, and maintain regenerative capacity over several injury-induced regeneration bouts.
However, upon injury, the stem cells become activated, divide to increase in number and then develop into new muscle fibers to replace those that were damaged.